Dear translators, we're migrating the GNU Health development environment to Codeberg! During this time, the translation will be locked until the migration process is done. Please follow the status on our official Mastodon account: https://mastodon.social/@gnuhealth

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Translation status

12,880 Strings 0%
Browse Translate
366,034 Words 0%
2,774,541 Characters 0%

Strings status

Strings Words Characters
12,880 366,034 2,774,541
All strings Browse Translate Zen
12,880 366,034 2,774,541
Unfinished strings Browse Translate Zen
12,820 365,863 2,773,040
Untranslated strings Browse Translate Zen
60 171 1,501
Strings marked for edit Browse Translate Zen
12,880 366,034 2,774,541
Unfinished strings without suggestions Browse Translate Zen

Other components

Component Translated Unfinished Unfinished words Unfinished characters Untranslated Checks Suggestions Comments
health GPL-3.0 78% 627 1,994 13,146 457 27 0 0
health_archives GPL-3.0 0 0 0 0 3 0 0
health_caldav GPL-3.0 3% 169 369 2,314 107 8 0 0
health_calendar GPL-3.0 80% 7 54 379 7 0 0 0
health_contact_tracing GPL-3.0 0% 30 87 587 16 0 0 0
health_crypto GPL-3.0 78% 14 79 506 11 0 0 0
health_crypto_lab GPL-3.0 0% 35 164 922 18 0 0 0
health_dentistry GPL-3.0 0% 1,016 1,159 5,783 693 0 0 0
health_disability GPL-3.0 0% 481 1,465 10,756 447 0 0 0
health_ems GPL-3.0 0% 202 518 3,321 143 0 0 0
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Overview

Project website www.gnuhealth.org
Project maintainers User avatar meanmicio
Translation license GNU General Public License v3.0 only
Translation process
  • Translations can be made directly.
  • Translation suggestions can be made.
  • Any authenticated user can contribute.
  • The translation uses bilingual files.
Source code repository https://hg.savannah.gnu.org/hgweb/health/
Repository branch default
Last remote commit Include README.rst in root page f5a6b7fabb4b
User avatar meanmicio authored a month ago
Last commit in Weblate Translated using Weblate (Swedish) f9cef6f9214d
Kristoffer Grundström authored a month ago
File mask tryton/health_genetics_uniprot/locale/*.po
Translation file Download tryton/health_genetics_uniprot/locale/ja_JP.po
Last change Jan. 17, 2024, 3:50 a.m.
Last author Feng Shu
3 weeks ago

String statistics

Strings percent Hosted strings Words percent Hosted words Characters percent Hosted characters
Total 12,880 366,034 2,774,541
Translated 0% 0 0% 0 0% 0
Needs editing 1% 60 1% 171 1% 1,501
Read-only 0% 0 0% 0 0% 0
Failing checks 0% 0 0% 0 0% 0
Strings with suggestions 0% 0 0% 0 0% 0
Untranslated strings 99% 12,820 99% 365,863 99% 2,773,040
Last 12 months Previous

Activity in last 12 months

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Apr May Jun Jul Aug Sep Oct Nov Dec Jan Feb Mar

Quick numbers

366 k
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12,880
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0
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Trends of last 30 days

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User avatar tumashu

Resource updated

  1. GNU Health
  2. health_genetics_uniprot
  3. Japanese
The “tryton/health_genetics_uniprot/locale/ja_JP.po” file was changed. 3 months ago
User avatar None

String added in the repository

  1. GNU Health
  2. health_genetics_uniprot
  3. Japanese
A form of non-syndromic, sensorineural hearing loss. Sensorineural hearing loss results from damage to the neural receptors of the inner ear, the nerve pathways to the brain, or the area of the brain that receives sound information. DFNA88 is characterized by postlingual, progressive and severe hearing loss with tinnitus.
 
3 months ago
User avatar None

String added in the repository

  1. GNU Health
  2. health_genetics_uniprot
  3. Japanese
A form of non-syndromic, sensorineural hearing loss. Sensorineural hearing loss results from damage to the neural receptors of the inner ear, the nerve pathways to the brain, or the area of the brain that receives sound information. DFNA87 is characterized by prelingual, profound sensorineural hearing loss with inner ear anomalies, including cochlear maldevelopment, absence of the osseous spiral lamina, and/or an enlarged vestibular aqueduct.
 
3 months ago
User avatar None

String added in the repository

  1. GNU Health
  2. health_genetics_uniprot
  3. Japanese
A form of non-syndromic, sensorineural hearing loss. Sensorineural hearing loss results from damage to the neural receptors of the inner ear, the nerve pathways to the brain, or the area of the brain that receives sound information. DFNA86 is characterized by progressive, bilateral hearing loss that is most predominant in the high frequencies, begins mildly during the fourth decade and gradually progresses to severe-to-profound deafness in the seventh and eighth decades. Affected subjects have tinnitus, while vestibular dysfunction or other clinical abnormalities are not present.
 
3 months ago
User avatar None

String added in the repository

  1. GNU Health
  2. health_genetics_uniprot
  3. Japanese
A congenital myopathy of variable severity, ranging from severe fetal akinesia to milder forms of muscle weakness. Most affected individuals show delayed motor development with generalized hypotonia and progressive axial and limb muscle weakness beginning soon after birth or in infancy. Additional features may include swallowing difficulties, external ophthalmoplegia, ptosis, high-arched palate, and respiratory insufficiency. Muscle biopsy shows variable morphologic abnormalities, including alveolar changes in the intermyofibrillar network, fiber size variability, focal disorganization, internal nuclei, and dilated sarcoplasmic reticulum and T-tubules. CMYP18 inheritance is autosomal dominant or recessive.
 
3 months ago
User avatar None

String added in the repository

  1. GNU Health
  2. health_genetics_uniprot
  3. Japanese
A form of paroxysmal kinesigenic dyskinesia, a neurologic condition characterized by recurrent and brief attacks of abnormal involuntary movements. These attacks can involve dystonic postures, chorea, or athetosis. EKD3 is an autosomal dominant form with incomplete penetrance and onset in late childhood or early adolescence. Symptoms are usually triggered by sudden movement or stress, and resolve in most patients in their early twenties or later.
 
3 months ago
User avatar None

String added in the repository

  1. GNU Health
  2. health_genetics_uniprot
  3. Japanese
A form of primary lymphedema, a disease characterized by swelling of body parts due to developmental anomalies and functional defects of the lymphatic system. Patients with lymphedema may suffer from recurrent local infections. LMPHM13 is an autosomal recessive form characterized by fetal onset of pleural and peritoneal effusions and the presence of moderate to severe non-immune hydrops fetalis that often resolves with age. Affected individuals show relatively normal growth and development, apart from mild ascites and hemangiomas. Most patients have congenital cardiac defects.
 
3 months ago
User avatar None

String added in the repository

  1. GNU Health
  2. health_genetics_uniprot
  3. Japanese
A form of cataract, an opacification of the crystalline lens of the eye that frequently results in visual impairment or blindness. Opacities vary in morphology, are often confined to a portion of the lens, and may be static or progressive. In general, the more posteriorly located and dense an opacity, the greater the impact on visual function. CTRCT50 is an autosomal dominant form characterized by early onset. Affected individuals may also exhibit high-tension glaucoma and variable anterior segment defects.
 
3 months ago
User avatar None

String added in the repository

  1. GNU Health
  2. health_genetics_uniprot
  3. Japanese
A form of hypomyelinating leukodystrophy, a group of heterogeneous disorders characterized by persistent deficit of myelin observed on brain imaging. HLD25 is an autosomal dominant form with onset in early infancy and characterized by nystagmus, hypotonia, and delayed global development. Most patients show gradual clinical improvement over time with resolution of the nystagmus in early childhood. Many achieve developmental milestones and may have normal cognition, although some affected individuals may have persistent neurologic deficits. Brain imaging shows hypomyelination that may also improve with time.
 
3 months ago
User avatar None

String added in the repository

  1. GNU Health
  2. health_genetics_uniprot
  3. Japanese
An autosomal recessive disorder characterized by global developmental delay, impaired intellectual development, absent speech, and behavioral abnormalities, including hyperactivity and attention deficit disorder. Affected individuals show failure to thrive with poor overall growth, and some have microcephaly. Additional features may include non-specific facial dysmorphism, hypotonia, and feeding difficulties.
 
3 months ago
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